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Health benefits of chewing gum? Who would have guessed? It true though. Actually there are 7 health benefits of chewing gum. This is great because gum is relatively cheap, can be chewed practically anywhere, and because of its sticky quality, can be discarded in more desecrate places that any other type of food or candy ever made. All kidding aside, the health benefits of chewing gum are real and shouldn’t be brushed off as silly. Read on and find out why.

1. It has been clinically proven that chewing a piece of sugar-free gum after eating a meal will help to significantly reduce the development of gingivitis and other gum diseases as well as tooth decay. Chewing gum stimulates the production of saliva. Saliva is the body’s natural defense against these unwanted oral conditions. Saliva neutralizes plaque acids, remineralizes tooth enamel which helps teeth stay strong and healthy, and washes away food particles left on the teeth and gums.

2. Research studies have link chewing gum with the ability to focus better, perform better and absorb and retain what you learn more easily, though how it works isn’t really known. A Study at the University of Northunbria in 2002 showed that a person’s cognitive abilities were dramatically increased when they chewed gum. Finding the link between chewing gum, and these heightened abilities, is still under investigation, but the results can’t be disputed.

3. If you want whiter teeth you might want to pop a piece of gum in your mouth rather than invest lots of money in a tooth whitening system or spend a mint to have your teeth whitened by a professional. There are numerous brands of gum that actually brighten the surface of your teeth while you chew them.

4. Chewing gum can help with digestive issues. As previously stated, chewing gum stimulates the production of saliva. Because saliva production causes you to swallow more frequently the chance of stomach acids travelling back up the throat, which can result in severe heartburn, are significantly reduced.

5. Many people don’t associate the act of chewing gum with weight loss. It’s true that chewing gum only burns about 11 calories, which doesn’t amount to much when it comes to the battle of the bulge, but it also helps to reduce your cravings for all those high-calorie snacks that pack on the pounds.

6. Chewing gum can help to relieve stress by releasing some of the energy that builds up in your body and causes stress. Stress is a real danger when it comes to good health. Many people underestimate the negative effects that stress has on the body. Be careful, though. Chewing gum in excess could lead to jaw troubles. Talk with your dentist to get some better insight regarding this.

7. It is thought that chewing gum contributes to a person’s overall good health. This makes sense when you take into consideration all the individual health benefits of chewing gum.

Maryellen Ward blogs about how to choose the masters in healthcare degree for you.

In the ferret, adrenal disease – or hyperadrenocorticism – most commonly occurs when a tumor or lesion on the adrenal glands causes an overproduction of the hormones produced by the adrenal glands. Adrenal disease is most common in ferrets over the age of three years, but it can affect ferrets as young as a year old. Adrenal disease is one of the most prevalent ferret diseases, and there is no definitive test for it. Ferret owners need to know as much as possible about it to be able to recognize it and help the ferret after he is diagnosed by the veterinarian.

CAUSES

While we don’t know for sure what exactly causes adrenal disease in ferrets, most theories point to two possible causes:

Spaying and neutering at a young age

Many large ferret breeders spay or neuter ferrets at the age of 5 or 6 weeks, which may cause the ferret’s body to overcompensate for the lack of normal sex hormones. Adrenal disease has occurred in ferrets that are spayed or neutered after they have reached sexual maturity, but it is not nearly as common.

Extended photoperiods

Most ferrets have been forced to adapt to their owner’s lifestyle and are exposed to at least four or five hours of artificial light in the evening in addition to the natural light during the day. This limits the time they spend in darkness, which decreases melatonin production. Too little melatonin results in overstimulation of the adrenal glands. Ferrets should have no less than 12 hours of complete darkness throughout a 24 hour period.

When a ferret develops adrenal disease, it is most likely the combination of a variety of factors. In addition to environmental factors, some ferrets are probably genetically predisposed to developing adrenal gland cancer.

SIGNS

Hair loss or hair thinning – at the base of the tail, on his feet, on his belly, in an obvious pattern, or in a patchy appearance – are classic signs of adrenal disease. There are multiple causes for hair loss in ferrets, but a ferret owner should always suspect adrenal disease and report this to his or her veterinarian, especially if the ferret is 3 years of age or older. However, while hair loss is the classic sign of adrenal disease, not all ferrets will have this sign.

Unlike other diseases, there is not a standard set of signs that a ferret with adrenal disease will always display. The range of signs that your ferret shows will depend on where the tumor is on his adrenal glands. The adrenal glands produce many hormones, so the location of the tumor will affect which hormones are overproduced. The types of hormones that are overproduced determines which signs you will see.

Here are the various signs of adrenal disease:

Hair loss, either in a symmetrical pattern or patchy with no apparent pattern *

Thinning hair

Loss of appetite

Lethargy

Papery thin or translucent looking skin, sometimes with sores from scratching

Excessive scratching and itchiness

Increase in musky body odor

Excessive grooming of self or other ferrets, including ear sucking

Sexual aggression and mating behavior in neutered males – with other ferrets, inanimate objects, etc.

Swollen vulva in spayed females

Difficulty urinating for males – this is a sign of an enlarged prostate, a condition usually associated with adrenal disease

Weakness in back legs – usually seen in advanced or extreme cases

Increased thirst, increased urination

Weight loss due to a decrease in muscle mass, but with a pot bellied appearance

* It is important to note that you may see partial or complete hair regrowth without treatment. This does not mean that your ferret is fine, it just means that the hormonal imbalances have balanced out again, probably due to a season change. The hormones will become unbalanced again, and hair loss will occur, usually more severe than before.

Always remember that you can see any combination of these signs – there is no set group of signs! Keep an eye on your ferrets as they grow older, watching for any symptoms or odd behavior. Something as seemingly innocuous as your ferret running from litter box to litter box trying to go to the bathroom should result in a trip to the veterinarian.

DIAGNOSING ADRENAL DISEASE

If your veterinarian has a lot of experience with ferrets or if your ferret has the traditional hair loss, he or she may be able to diagnose your ferret through clinical signs (symptoms).

Your veterinarian may decide to use the Adrenal Panel run by the University of Tennessee, often referred to as the “Tennessee Panel,” in cases where adrenal disease is suspected. This is a blood test that evaluates the levels of hormones and steroid production. The test is not always 100% correct, and has been known to result in false positives and false negatives, but it is the only blood test available to test for adrenal disease.

Radiographs (x-rays) and ultrasounds help determine whether or not adrenal disease is present, but can be misleading. Diseased adrenal glands can be normal in size and shape and, therefore, an x-ray or ultrasound may not raise any concerns. However, ultrasounds can be helpful in showing prostate enlargement (a complication of adrenal disease in male ferrets).

When it comes to diagnosis, if your ferret is displaying any of the obvious signs, your veterinarian will probably suggest beginning treatment rather than spending money on expensive tests and other diagnostic measures. Ferrets can suffer from adrenal disease for a long time before actually showing symptoms, so no matter how your veterinarian arrives at the diagnosis, it’s important to commence treatment immediately after the diagnosis has been made.

TREATMENT

Your veterinarian may opt to use any of the following options to treat your ferret’s adrenal disease:

Surgery

This is the most common treatment for adrenal disease, and is the only way to completely remove the tumor. It is also the most recommended treatment by most veterinarians. If your veterinarian seems at all hesitant about the surgery or does not perform surgeries, ask for a recommendation to a more experienced ferret veterinarian.

If your ferret has already had surgery to remove one gland and adrenal tumors develop in the remaining gland, your veterinarian may decide that surgery is not the best choice. Ferrets that have both glands removed may develop Addison’s disease, which is a severe or complete deficiency of the hormones made in the adrenal glands. Your veterinarian will probably want to check your ferret’s cortisol (a hormone produced by the adrenal glands) levels within three days of the surgery.

Though surgery is the only option that could completely rid your ferret of adrenal disease, it is important for you to know that there are risks involved with surgery.

If the tumor is on the right adrenal gland, your veterinarian may choose not to remove it. The right adrenal gland is directly adjacent to the vena cava, the largest vein in the body, and there could be serious complications.

If any microscopic piece of affected adrenal tissue remains, the tumor can grow back either on the same side, or the opposite side.

There is always a chance that the ferret may not make it through the surgery or through the recovery period in the first few days after the procedure is done.

Discuss all of the risks extensively with your veterinarian before making any decisions. Some ferrets are just not good surgical candidates, either due to age, health or both, and with these ferrets your veterinarian will probably want to go the medical treatment route.

Medical

Your veterinarian may suggest medical treatment options in cases where surgery cannot be performed, either due to the health of the ferret or other issues. These options include:

Lupron Depot Injections

Lupron Depot may be prescribed at 1 month, 3
month, or 4 month intervals. “Depot” refers to the fact that the entire dosage is released over the given time period. This means that your ferret will need to get this shot at the appropriate time for the rest of his life. Lupron may shrink the tumor, but in most cases, it only affects the symptoms.

Even if all symptoms cease, Lupron must continue to be administered. Lupron is a synthetic version of GnRH (gonadotropin release hormone), and it works by desensitizing the pituitary gland, which stops the production of the hormones that are overstimulating the adrenal glands. So if the Lupron shots are stopped, the pituitary gland resumes its normal function, and all of the problems start again.

If your veterinarian recommends doing surgery, but you can’t do it right away for whatever reason, some veterinarians may recommend that the ferret receive Lupron in the meantime. This may prevent the disease from progressing any further and, at the very least, may make your ferret more comfortable by alleviating some of the symptoms.

Melatonin Implants

Melatonin is a natural hormone that serves many functions in your ferret’s body, one of which is to inhibit the release of GnRH (gonadotropin releasing hormone). Less GnRH means that the pituitary gland releases fewer hormones, which means that the adrenal glands are stimulated less.

Melatonin can be used in oral (liquid or pill) or implant form. While you can use oral melatonin, the success or failure of it depends on the time of day it is given. It needs to be administered exactly 7 – 9 hours after sunrise to mimic the natural release of melatonin. If given at this time every single day, oral melatonin is extremely effective. Unfortunately many ferret owners are not home during this time. A more convenient way to administer melatonin effectively is to use Ferretonin, a melatonin implant. Implants last about 3 – 4 months, and provide a steady level of melatonin over that time period.

Melatonin implants alone cannot be used to treat adrenal disease, and the best results are seen if Melatonin implants are used in conjunction with Lupron Depot.

Medications to avoid

Lysodren (mitotane) was formerly used as a common treatment for adrenal disease, but studies have shown that Lysodren doesn’t stop the stimulation of the adrenal glands and causes low blood sugar. This is a concern, as insulinoma and adrenal disease are often seen together, and ferrets with insulinoma struggle with low blood sugar already. Overdosages or incorrect dosages of Lysodren were also known to cause Addison’s Disease.

Nizoral (Ketoconazole) is used to treat Cushing’s disease (hyperadrenocorticism) in dogs, which is completely different from adrenal disease (also hyperadrenocorticism) in ferrets. Though they are the same disease, the same drugs cannot be used to treat both.

Vetoryl (Trilostane) is also used in dogs with Cushing’s Disease, and increases the level of a hormone that is already quite high in ferrets with adrenal disease. Giving this to a ferret with adrenal disease would make the problem worse.

Nolvadex (Tamoxifen) is an anti-estrogen medicine in humans. However, it actually has estrogen-like effects in ferrets, which would have a negative effect on many ferrets with adrenal disease.

PREVENTING ADRENAL DISEASE

Unfortunately, there are no proven ways to prevent adrenal disease. It is currently thought that the early neutering/spaying that is done by large scale breeders directly contributes to adrenal disease, and in most cases, the ferrets that you get will already have had this surgery. If they are intact, it is recommended that you wait until they are at least 6 months of age before getting them spayed or neutered. Adrenal disease is still seen in ferrets who are spayed or neutered after reaching sexual maturity, but it is not as prevalent.

Recent studies have shown that light cycles also contribute to the development of adrenal disease. Melatonin, as mentioned above, regulates the release of GnRH. GnRH affects how much the adrenal glands are stimulated. Melatonin is produced when the ferret is in total darkness. Most of our ferrets live in the same environment we do – natural daylight during the day, and several hours of artificial light in the evenings. This obviously shortens the amount of time the ferret is in darkness, thereby decreasing the production of melatonin. Less melatonin means more GnRH is released, which then overstimulates the adrenal glands leading to adrenal disease. A ferret’s optimal light cycle is about eight hours of light and sixteen hours of total darkness each day. It is thought that this will allow your ferret to produce the most melatonin possible. There are some ferret supplies, such as cage covers and certain types of fabric bedding, available to help you create the necessary photoperiods.

IN SUMMARY

It’s important to add one more thought here – adrenal disease is very treatable in most cases. Not treating adrenal disease is condemning your ferret to die, when this doesn’t have to be the case. If you see signs of adrenal disease in your ferret, please get him checked out as soon as possible. Ferrets can live long and happy lives after they are treated for adrenal disease, and I know we all want that for our fuzzies!

Misconceptions about cardiovascular diseases — heart attacks, stroke and high blood pressure — have existed for many years and have in effect become myths. Most of them stem from factual observations during the early phase of the current global epidemic and have become deeply rooted in the minds of policymakers, health professionals and the public alike. Since these misconceptions adversely influence the allocation of resources and undermine actions to prevent and control cardiovascular diseases, they need to be firmly squashed.

Myth 1: Heart disease is a problem of developed countries

Every year, cardiovascular diseases cause around 15 million deaths in the world (30% of all deaths), and of these about two-thirds occur in developing countries. So the absolute number of deaths from these causes is twice as high in developing countries as in the industrialized world. Over twice as many deaths from stroke occur in developing countries as in industrialized countries; and the numbers of deaths due to heart attacks are equal in poor and rich countries. It is estimated that in China and India combined, which account for half the population of the developing world, between five and six million deaths are caused each year by cardiovascular diseases.

Myth 2: Heart disease is a problem of the rich

All societies include “early adopters” and “late adopters” of lifestyle changes. Early in the heart disease epidemic, affluent people in developing countries had the means and the opportunity to adopt new lifestyles, involving behaviour such as choosing foods rich in fat and calories, buying cars and using tobacco. Since these goods have become affordable for mass consumption “unhealthy” behaviour of this kind has become common across all social classes. Today, affluent people, especially the urban rich, have better access to health information concerning risk factors in the media and they also possess the means to modify their behaviour in favour of a healthier lifestyle (healthy diets, leisure-time physical activity, abstinence from tobacco). They constitute the “early adopters”, while the urban poor and rural communities — with limited access to information and little time or money for “healthy foods” and “fitness clubs” — lag behind. As a result, risky behaviour develops, and risk factors increase.

Recent studies from Latin America and South-East Asia, where coronary heart disease is particularly common, indicate that many coronary risk factors are more prevalent among those with lower socioeconomic standing and that the poor are, indeed, at higher risk of heart attacks.

In industrialized countries too, where the epidemic began among the urban rich, though some decades earlier than in the developing world, cardiovascular diseases are now more common in the relatively poor. When the worldwide heart disease epidemic fully develops, the poorest countries and the poorest people within society will be the worst affected.

Myth 3: Heart disease is mostly a man’s disease

While coronary heart disease is, in general, less common in pre-menopausal women than in men, in many parts of the world it is the most common cause of death in women, even those aged under 65. Heart disease, as well as its risk factors, varies to a surprising degree between populations. For example, women aged 35-64 years in Glasgow, Scotland, and in Belfast, Northern Ireland, have higher heart attack rates than men in some parts of southern Europe, according to a recent WHO study on trends in cardiovascular diseases (the WHO MONICA Project).

Hypertension and stroke are also major problems that affect women. Given the longer life expectancy of women, they contribute increasingly to cardiovascular deaths and disability after the sixth decade. The result is that, over their entire lifespan, women and men are equally affected by heart attacks and stroke — a fact that has long been neglected by doctors and health professionals, and by women themselves. Furthermore, pregnancy-associated hypertension is an important health problem in the developing world, where it is the major cause of premature birth and perinatal death, and is also responsible for up to one-third of all maternal deaths.

Myth 4: Heart disease is a problem of old age

Atherosclerotic cardiovascular diseases (coronary heart disease and stroke) and hypertension increase with age. But research in industrialized countries shows that about one-third of heart attacks and one-quarter of strokes occur in people below the age of 65. Many of the deaths due to cardiovascular diseases also occur early, one-quarter of them below the age of 70. In the developing world, the situation is even more marked: up to half of all deaths attributable to heart diseases occur in persons younger than 70; and a great number of working-age adults suffer from these diseases. This has an enormous impact on the economic situation of individuals and families as well as on society as a whole, and hampers efforts to alleviate poverty.

Myth 5: Heart disease is not susceptible to community action

The predominant factors contributing to the risk of cardiovascular diseases appear to be acquired, and to be lifestyle-related rather than genetic. Risk factors can be modified within a “healthy environment” that supports appropriate lifestyle practices, and most cardiovascular diseases are preventable. The prevention of heart diseases in individuals calls for the active promotion of health in populations.

Programmes that combine community mobilization with governmental regulation through taxation, legislation and pricing policies have proved to be effective in controlling tobacco and encouraging healthier diets in numerous industrialized countries. From these experiences, it is clear that community, national and even global action are key elements in combating the advancing epidemic of cardiovascular diseases in the developing world. Community mobilization can best be attained through educating the public, patients, professionals and policymakers, based on the advice of health professionals.

Myth 6: Heart disease is no longer a public health issue

There is a widespread mistaken belief that the total burden of cardiovascular diseases is diminishing. Despite declining mortality, heart disease remains the dominant public health problem in industrialized countries. Eastern European countries are at present experiencing the highest mortality rates due to cardiovascular diseases. A major cause for concern is the projected rise of these diseases in developing countries in the next century. It is predicted that by 2020 the number of deaths due to heart attacks and stroke in the developing world will have doubled as compared with 1990.

The reasons for this anticipated acceleration of the epidemic are increasing life expectancy related to a decline in infant mortality, unhealthy lifestyle changes related to industrialization and urbanization, and longer periods of exposure to the risk factors of heart disease because of improved socioeconomic conditions.

The public health consequences of an uncontrolled epidemic of cardiovascular diseases in the developing world would be disastrous. Not only would millions of productive years of life be lost, but the high costs of technology-intensive management of these diseases would impose a heavy financial burden on affected individuals, their families and society as a whole. The global epidemic needs a global response now, in the form of an international effort to create awareness and stimulate action in all countries and all sectors of society.